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Many athletes use anabolic-androgenic steroids (aas) for physical enhancement but the magnitude of these gains and associated adverse effects has not been rigorously quantified. This is true even though a body of research indicates a variety of significant negative health effects and harms associated with asex hormones and their related drugs as well as the risk of abuse and misuse ( 1 , 2 ). A recent study of professional male athletes ( 3 ) evaluated the performance differences between the effects of testosterone (TS) – a natural anabolic steroid – and dutasteride on endurance and power during a single bout of moderate-intensity exercise. Athletes on the dutasteride group significantly increased their endurance and power performance. No significant alterations in body composition were observed. In contrast, the testosterone group significantly (P <0.05) increased their 1-RM bench press without any significant alterations in body composition parameters. The results are of interest because although a considerable amount of data exists, its reliability is uncertain and it is not known whether the differences observed were due to differences in performance parameters among the two groups and to the use of different dutasteride formulations. A recent review of the literature and the available data suggested that a positive body fat percentage, body fat percentage, skinfold thickness and lean body mass of the asexed subjects reported in the study were significantly correlated with changes in aerobic endurance performance after taking a dutasteride ( 4 - 7 ). In the present paper, we attempt to elucidate the physiological mechanisms of the positive (i.e., increased endurance and power) and negative (i.e., increased fat and body fat percentage) effects of aas. Furthermore, this study investigated the possible differences in their effects caused by the formulation of asexasthenol in both testosterone and dutasteride groups.

We examined in detail the hormonal effects of asexasthenol in a 1 or 1.5-mg/kg body weight dutasteride administered in combination (in random order within a 5-min intertrial interval) to healthy subjects in a randomized crossover design in a double-blind, placebo-controlled, crossover manner. In the second and third phases, we assessed the effects of asexasthenol on plasma parameters: asexaporin (a muscle lipid metabolite with a low affinity for testosterone); glycohemoglobin (a metabolic marker of high-intensity exercise); and plasma (lipid) and erythrocyte (protein) concentrations of luteinizing hormone (LH) and follicle-stimulating hormone (FSH).

To establish a true, positive anabolic response, we used a power-load protocol using a

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